Ukuhlolwa kwe-chloride - igazi
I-chloride uhlobo lwe-electrolyte. Isebenza namanye ama-electrolyte afana ne-potassium, i-sodium, ne-carbon dioxide (CO2). Lezi zinto zisiza ukugcina ibhalansi elifanele loketshezi lomzimba futhi zigcine i-acid-base balance yomzimba.
Le ndatshana imayelana nokuhlolwa kwelabhorethri okusetshenziselwa ukukala inani le-chloride engxenyeni ewuketshezi (i-serum) yegazi.
Kudingeka isampula yegazi. Isikhathi esiningi igazi likhishwa emthanjeni ongaphakathi endololwaneni noma ngemuva kwesandla.
Imithi eminingi ingaphazamisa imiphumela yokuhlolwa kwegazi.
- Umhlinzeki wakho wezokunakekelwa kwempilo uzokutshela uma kudingeka uyeke ukuthatha noma yimiphi imithi ngaphambi kokwenza lokhu kuhlolwa.
- UNGAYIMISI noma ushintshe imithi yakho ngaphandle kokukhuluma nomhlinzeki wakho kuqala.
Ungaba nalolu vivinyo uma unezimpawu zokuthi izinga lomzimba womzimba wakho noma ibhalansi ye-acid-base iyaphazamiseka.
Lokhu kuhlolwa kuvame uku-odwa ngokuhlolwa kwegazi, okufana nephaneli eyisisekelo noma ephelele yemetabolic.
Ububanzi obujwayelekile bungu-96 kuya ku-106 milliequivalents ilitha ngalinye (mEq / L) noma ama-millimole angama-96 kuye kwayi-106 ngelitha (millimol / L).
Ububanzi bamanani ajwayelekile bungahluka kancane phakathi kwamalabhorethri ahlukene. Khuluma nomhlinzeki wakho mayelana nencazelo yemiphumela yakho ethile yokuhlolwa.
Isibonelo esingenhla sikhombisa ibanga lesilinganiso elijwayelekile lemiphumela yalezi zivivinyo. Amanye ama-laboratories asebenzisa izilinganiso ezahlukahlukene noma angahlola izinhlobo ezahlukene.
Izinga elikhulu kune-evamile le-chloride libizwa nge-hyperchloremia. Kungahle kube ngenxa:
- Isifo i-Addison
- I-Carbonic anhydrase inhibitors (esetshenziselwa ukwelapha i-glaucoma)
- Uhudo
- I-Metabolic acidosis
- I-alkalosis yokuphefumula (inxephezelwe)
- I-acidosis tubular yezinso
Izinga eliphansi kunejwayelekile le-chloride libizwa nge-hypochloremia. Kungahle kube ngenxa:
- Isifo seBartter
- Kushe
- Ukwehluleka kwenhliziyo yokubopha
- Ukuphela kwamanzi emzimbeni
- Ukujuluka okweqile
- I-Hyperaldosteronism
- I-alkalosis yomzimba
- I-acidosis yokuphefumula (inxephezelwe)
- I-Syndrome ye-diuretic secretion engafanele (SIADH)
- Ukuhlanza
Lokhu kuhlolwa kungenziwa futhi ukusiza ukukhipha umthetho noma ukuxilonga:
- Ama-endocrine neoplasia amaningi (AMADODA) II
- I-hyperparathyroidism eyinhloko
Ukuhlolwa kwe-Serum chloride
- Ukuhlolwa kwegazi
IGiavarina D.I-biochemistry yegazi: ukukala ama-electrolyte amakhulu e-plasma. Ku: Ronco C, Bellomo R, Kellum JA, Ricci Z, abahleli. Ukunakekelwa Okubalulekile I-Nephrology. 3rd ed. IPhiladelphia, PA: Elsevier; 2019: isahluko 54.
Seifter JR. Izinkinga ze-acid-base. Ku: IGoldman L, iSchafer AI, ama-eds. Imithi yeGoldman-Cecil. Umhla ka-25. IPhiladelphia, PA: Elsevier Saunders; 2016: isahluko 118.
UTolwani AJ, Saha MK, Wille KM. I-Metabolic acidosis ne-alkalosis. Ku: Vincent JL, Abraham E, Moore FA, Kochanek PM, Fink MP, ama-eds. Incwadi Yokunakekelwa Okubalulekile. Umhla wesi-7. IPhiladelphia, PA: Elsevier; I-2017: isahluko 104.