Umlobi: Tamara Smith
Usuku Lokudalwa: 20 Ujanuwari 2021
Ukuvuselela Usuku: 3 Epreli 2025
Anonim
I-hypertrophic cardiomyopathy: kuyini, izimpawu, izimbangela nokwelashwa - Impilo
I-hypertrophic cardiomyopathy: kuyini, izimpawu, izimbangela nokwelashwa - Impilo

-Delile

I-hypertrophic cardiomyopathy yisifo esibi esiholela ekwandeni kobukhulu besicubu senhliziyo, sisenze siqine futhi sibe nobunzima obukhulu bokumpompa igazi, okungaholela ekufeni.

Yize i-hypertrophic cardiomyopathy ingenakho ukwelashwa, ukwelashwa kusiza ekunciphiseni izimpawu futhi kuvimbele inkinga ukuthi ibe yimbi kakhulu, kuvimbele izinkinga ezifana ne-atrial fibrillation ngisho nokuboshwa kwenhliziyo, ngokwesibonelo.

Bona izimpawu eziyi-12 ezingakhombisa izinkinga zenhliziyo.

Izimpawu eziyinhloko

Ezimweni eziningi, i-hypertrophic cardiomyopathy ayibonisi zimpawu noma zimpawu, futhi ivame ukukhonjwa ekuhlolweni kwenhliziyo okujwayelekile. Noma kunjalo, abanye abantu bangathola:

  • Ukuzwa ukuphefumula okuncane, ikakhulukazi lapho wenza imizamo yomzimba;
  • Ubuhlungu besifuba, ikakhulukazi ngesikhathi sokuzivocavoca umzimba;
  • Ukushaya kancane noma ukuzwa kwenhliziyo okusheshayo;

Ngakho-ke, lapho kuvela noma yiziphi zalezi zimpawu, kungakuhle uye kudokotela ukuyokwenza izivivinyo ezidingekayo, njenge-echocardiography noma i-X-ray yesifuba, esiza ukukhomba inkinga bese uqala ukwelashwa okufanele.


Imvamisa, ngokuguga nokuqina kwenhliziyo, kujwayelekile futhi ukuthi kuphakame umfutho wegazi ophakeme kanye nama-arrhythmias, ngenxa yokuguqulwa kwezimpawu zikagesi ezisemisipha yenhliziyo.

Izimbangela ezingaba khona

I-hypertrophic cardiomyopathy imvamisa ibangelwa ukuguqulwa kofuzo okubanga ukukhula okweqile kwemisipha yenhliziyo, eba mkhulu kunokujwayelekile.

Ushintsho oludala lesi sifo lungadlulela kubazali luye ezinganeni, ngamathuba angama-50% okuthi izingane zizalwe zinenkinga, noma ngabe lesi sifo sithinta umzali oyedwa kuphela.

Yelashwa kanjani

Ngakho-ke, udokotela wenhliziyo uvame ukuqala ukwelashwa esebenzisa amakhambi afana nalawa:

  • Amakhambi okuphumuza inhliziyo, njengeMetoprolol noma iVerapamil: kunciphisa ukucindezeleka emisipheni yenhliziyo futhi kwehlise izinga lokushaya kwenhliziyo, okuvumela ukupompelwa kwegazi ngempumelelo;
  • Izindlela zokulawula ukushaya kwenhliziyo, njenge-Amiodarone noma iDisopyramide: gcina izinga lokushaya kwenhliziyo lingaguquguquki, ugweme ukusebenza ngokweqile yinhliziyo;
  • Ama-anticoagulants, njengeWarfarin noma iDabigatran: asetshenziswa lapho kune-fibrillation ye-atrial, ukuvimbela ukwakheka kwamahlule angadala i-infarction noma isifo sohlangothi;

Kodwa-ke, lapho ukusetshenziswa kwale mithi kungakwazi ukunciphisa izimpawu, udokotela angasebenzisa ukuhlinzwa ukususa ucezu lomsipha wenhliziyo ohlukanisa ama-ventricles amabili enhliziyweni, enza lula ukudlula kwegazi futhi anciphise umzamo inhliziyo.


Ezimweni ezinzima kakhulu, lapho kunengozi enkulu yokuboshwa kwenhliziyo ngenxa ye-arrhythmia, kungadingeka ukufaka umshini wokushaya inhliziyo enhliziyweni, okhiqiza amandla kagesi akwazi ukulawula isigqi senhliziyo. Ukuqonda kangcono ukuthi i-pacemaker isebenza kanjani.

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